The treatment is based on a protein called biglycan.

.. In mice, the treatment is based on a protein called biglycan, was by 50 % type to the muscle damage, probably the cause probably the cause the muscle to be worsening boys with Duchenne muscular dystrophy (DMD ,, said Justin Fallon, professor of neuroscience, who led the research. Acts in all 3,500 boys Affected are an incurable an incurable fate in the scar tissue and fat almost every skeletal muscle in their body carries. Patients inevitably die, typically by the time they are in their 20s. – At this point we can not say how effectively is biglycan in humans, but the results from the mouse studies are encouraging, said Fallon, who is with the Brown – affiliated Institute for Brain Research. We seek an effective treatment, the boys are a better quality of life for years.

Joel Braunstein, one of Tivorsan founder and current CEO, the company will raise the money and hire the technical staff to see biglycan by the through the development of drugs and government regulatory agencies review and approval processes. – We have a challenging road ahead of us, but we strongly work towards examining the safety and efficacy of biglycan committed humans, said Braunstein, a physician and principal at LifeTech Research in Baltimore. We are deeply the need and urgency for a new therapy recognized in DMD. If biglycan proves tested safe and effective in humans, a team of a relief for thousands of boys and their families throughout the world. .– ‘By earlier immunization, we have given in principle at has these mice intended lifetime protection for a disease they were,’said the study study leader, Martin Kast, Professor for Molecular Microbiology & Immunology and Obstetrics and Gynecology to the Norris Comprehensive Cancer centers. ‘This has never done before, could with further research to defend a change of paradigm for the treatment of human prostate cancer. ‘.. Researchers at the University of Southern California are a prostate cancer vaccine which predestined development of cancer to 90 % of young mouse developing the disease to develop the disease prevented from developed.

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